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Home Page >Online Product Catalog > Primary Antibodies > Hyperpolarization-activated Cyclic Nucleotide-gated channel 1-4 (HCN1-4) > Human Hyperpolarization-activated Cyclic Nucleotide-gated channel 2 (HCN2) Control/blocking peptide (Peptide)

Human Hyperpolarization-activated Cyclic Nucleotide-gated channel 2 (HCN2) Control/blocking peptide

Catalog# Product Description Product Type Antibody Type Size Price
HCN21-P Human Hyperpolarization-activated Cyclic Nucleotide-gated channel 2 (HCN2) Control/blocking peptide Peptide 100 ug $95.00



SugApplications
Control or blocking peptides are recommended for ELISA plate coating for direct ELISA testing of antibodies or competitive ELISA, and antibody blocking or neutralization.

SpeciesReactivity
Human/Mouse/Rat

Form
Antigen/blocking Peptide

Antigen
15-aa peptide from human HCN2 (1); Designation (#HCN21-P, control/blocking peptide) conjugated to KLH; epitope location ~ N-terminal, Cytoplasmic domain

SpCrossreactivity
The HCN21-P peptide is 100% conserved in human, mouse and rat HCN2. No significant sequence homology of HCN21-P is seen with other HCNs or any other protein. Antibody reactivity in various species is not known

For research use only, not for diagnostic or therapeutic use.

Data Sheets
HCN21-S-A-P.pdf

MSDS

General Information

HCN2, also known as Brain cyclic nucleotide channel gated 2, BCNG2 or HAC-1 (rat 834 aa, human 889-aa, chromosome 19p13.3), is primarily expressed in brain. In mouse, HCN1 is mainly expressed in brain (olfactory bulb and cerebral cortex, hippocampus, tha

Citations
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    showing 1 - 2 of 2  
ADI Product Used Authors, Year, Journal, Title Technique used Comments
HCN-3 ab # HCN31-a Wells JE  2007  International Endodontic Journal., 40, 715–721, 2007  Hyperpolarization-activated channels in trigeminal ganglia innervating healthy and pulp-exposed teeth &n   ab used @1:1000-1:250; Rat trigeminal ganglia
HCN-3 Ab #HCN31-A Strauss U  2004  Eur. J. Neuros.19, 11, 3048-3058  An impaired neocortical I h is associated with enhanced excitability and absence epilepsy &n   rat brain



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